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thinness gene

01.06.2020

The genetic sequence of ALK (anaplastic lymphomakinase) turned out to be the thinness gene, which scientists have been looking for for quite some time. It is ALK that plays a key role in maintaining harmony.

European scientists, led by Dr. Josef Penninger from the Institute of Natural Sciences, compared the genetic parameters of 47 thousand slender people, and found that they are united by a special mutation in the ALK gene. Until now, scientists have not fully understood what exactly anaplastic lymphomakinase does in the human body - existing studies have shown that the gene may only be a factor in the development of certain types of cancerous tumors.

"Our study shows that the ALK gene is active in brain cells, where it is responsible for regulating metabolism. It is, quite simply, a regulator of energy expenditure," said Michael Orthofer from the Institute of Molecular Biology in Vienna, one of the lead authors of the project.

The scientists deactivated the ALK gene in model mice and observed how rodents that ate the same amount of food as animals from the control group gained markedly less weight in general and fat in particular. The authors of the work are confident that in the foreseeable future anti-obesity drugs will be created that directly affect the synthesis of ALK.

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